Adi Zheng and Christian Widmann
«HDLs, as SAA carrier, can hamper the latter from exerting its pro-inflammatory activities; in return, SAA, as an apolipoprotein, can change the characteristics of HDL particles. Understanding the dual/mutual relationship between the respective functions of SAA and HDLs will help define their physiologic roles as well as their contribution to the pathophysiology of atherosclerosis.”